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    Immune and Inflammatory Response after Cardiopulmonary Bypass: How to avoid adverse outcomes

    Cardiopulmonary Bypass (CPB) activates the immune system, resulting in a systemic inflammatory response. Downstream consequences have a multi-organ system impact, and, if severe enough, they may result in significant morbidity or mortality after cardiac surgery. Dr. Kristin C. Greathouse describes the serious consequences and the possibilities of prevention or therapy (in the new textbook "Extracorporeal Circulation").

     

    EXTRACORPOREAL CIRCULATION In Theory and Practice

    Importantly , the activation of the immune system and subsequent inflammatory response is simultaneously countered by a compensatory anti-inflammatory response , which serves to suppress the inflammatory response to augment return to immunological homeostasis. There has been increasing understanding that extremes of either an unbridled inflammatory response or persistent suppression of immune responses after cardiac surgery are associated with adverse outcomes. The systemic inflammatory response is strongly implicated in cardiac, renal, pulmonary, hematologic, and neurological dysfunction in the postoperative period.

    Greathouse describes extensively the approaches to reduce inflammation and improve immune function post Cardiopulmonary Bypass:

    Pump mechanisms: Mini extracorporeal circuits, biocompatible coating, modified ultrafiltration, leucocyte reducing filters 

    Pharmacologic mechanisms: Steroids, Serine Protease Inhibitors, other

    The most promising interventions are multi-modal ones (e.g. pharmacologic combined with mechanical strategies), those targeting multiple inflammatory pathways.

     

    Rudolf Tschaut et al. (Eds.)
    Extracorporeal Circulation in Theory and Practice.
    Pabst, 740 pages. Hardcover ISBN 978-3-95853-545-9. eBook ISBN 978-3-95853-546-6

     

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